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Therefore, our objective was to explore the interplay between two key players in BD disease pathophysiology: mitochondrial dysfunction and inflammation

Currently used diagnostic biomarker methods, such as cerebrospinal fluid (CSF) analysis and positron emission tomography (PET) imaging, provide valuable information but are invasive, expensive and not suitable as a front-line screener 5,7

Next, we isolated activated ILC2s from the lungs of IL-33 challenged mice, subsequently treating them with morpholinos to block the translation of NRF2 whilst treating them with melatonin ( Figures 7A, G )

Rats were administered porcine type II collagen and treated orally with CaME (50, 150, and 250 mg/kg/day) for 15 days, and the outcomes were measured