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Considerable progress has been made toward developing new drugs for ferroptosis, utilizing the well-known targets of the pathological conditions (160, 161)

Thus, copper bound to the mitogen-activated protein kinase kinase 1 (MEK1) enhances its phosphorylation, thereby promoting tumorigenesis [4]

These findings indicate that inflammatory conditions associated with the overactivation of NF-B may misalign the molecular clock, thereby altering the transcription of several target genes and contributing to metabolic inflammation through direct transcriptional reprogramming

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