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Thus, in this current study, the significant increase in serum ROS could have been due to the autoxidation of hemoglobin and activation of nicotinamide adenine dinucleotide phosphate oxidase in the MSG-administered groups, leading to the generation of superoxide radicals and their dismutation to hydrogen peroxides within the RBC (Wang and Zennadi, 2020)

Furthermore, although the physiological changes studied (mitochondrial dysfunction and abnormal redox metabolism) are associated with ASD, the biological and clinical significance of these changes in relation to other neurodevelopmental disorders and diseases have not been well studied, so their significance is not entirely clear at this time

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The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice and beta(3)-AR knockout mice