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However, these reactions invariably result in the generation of reactive oxygen species (ROS) compounds that are unstable by having final layer of electrons unpaired and that, in trying to stabilize itself sequester electrons from other biomolecules, making them also destabilizes and, therefore, is no longer able to perform their duties properly, thus altering the homeostasis and, ultimately, causing cell death

Studies using LPS and MPTP mouse models have reported similar findings, with pharmacologic activators of mitophagy causing reduced microglial activation and improved DAN viability [112, 113]

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