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The active enzyme in the oxidized state works as a homodimer linked by two intermolecular disulphide bonds between specific cysteines (Hothorn et al., 2004

4gj), and YM-53601 still inhibited ferroptosis in FDFT1 null cells (Supplementary Fig

In tolerance experiments with high doses of LPS-induced sepsis, changes in histone H3K4me3, H3K27me2, and histone methyltransferase complexes (102, 151, 152) can downregulate the genes encoding inflammatory factors while upregulating the anti-inflammatory factor genes (153, 154)

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