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However, under defined pathophysiological stresses, including inflammatory cues, metal overload, cysteine deprivation, or drug exposure, GSH oxidation and thiol chemistry may become coupled to metal redox cycling and mitochondrial damage amplification, thereby increasing susceptibility to forms of regulated cell death such as ferroptosis and cuproptosis

Whether this insulin resistance contributes to sodium retention or hypertension in SS rats is not clear

Am J Cancer Res 12(8):3561 Pandey P et al (2024) Review to elucidate the correlation between Cuproptosis-related genes and immune infiltration for enhancing the detection and treatment of cervical cancer