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doi: 10.1093/genetics/156.3.1349 68 PerottiM

Findings include: Accelerated wound closure rates in multiple animal models across all regenerative components KPV-mediated inflammation control: Reduced inflammatory cell infiltration (leukocytes, mast cells) Enhanced re-epithelialization and granulation tissue formation Improved collagen deposition and organization in healing wounds Smaller scar formation demonstrated in KPV studies, complementing tissue repair components Antimicrobial effects from KPV against S

It has been found that inhibition of the PLC1 isoenzyme leads to a decrease in intracellular Ca 2+ ion concentration and nuclear factor of activated T cells (NFAT) activity, as well as hyperactivation of mitogen-activated protein kinase (MAPK) protein kinase and inactivation of Ras-homolog of protein Ras homolog family member A (RhoA) in normal human epidermal keratinocytes (NHEK), which consequently may lead to a decrease in both ROS generation and inflammatory state [79]