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Similarly, PNPLA3 I148M promotes hepatosteatosis through ABHD5-dependent inhibition of PNPLA2 (75), suggesting that small molecules that target the PNPLA3 I148M/ABHD5 interaction could be potential therapeutics for FLD and diabetes

The high-resolution data allowed us to build unmodeled sequences, such as the N terminus of Rpn1, which is sandwiched between the toroidal domain of Rpn1 and Rpt1