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For the safety analysis, all patients randomized were considered

Imbalance in mitochondrial fission and fusion is a key trigger for Parkinsons disease, and defects in mitochondrial dynamics limit mitochondrial movement, leading to a reduction in ATP, excessive oxidative stress, and mtDNA deficiency, which ultimately leads to cell death (Wu et al., 2017), with a substantial body of evidence demonstrating the involvement of -Syn, DJ-1, LRRK2, PINK1, and Parkin in the regulation of mitochondrial dynamics and homeostasis through the Drp1 pathway (ODonnell et al., 2014

On the other hand, several drugs can deplete pyridoxine or heighten its requirement