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Downregulation of apoptosis-associated tyrosine kinase promotes iron accumulation via enhanced endosome recycling, sensitizing EGFR-TKI-resistant cells to ferroptosis inducers such as RSL3 (128)

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NO produced by eNOS improves ulcer healing by increasing vasodilation, inducing angiogenesis, scavenging the toxic-free radicals, and reducing leukocyte infiltration, resulting in epithelial tissue integrity restoration and enhanced mucous secretion ( FIGURE 5 3.5 Effect of PGG on Immunoexpression of Inducible Nitric Oxide Synthase Nitric oxide produced by iNOS plays a role in ulcer formation through the production of cytotoxic peroxidefree radicals (Pan et al., 2005)

Psychiatry Clin Neurosci 2012;66:514-7