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In addition, due to the inability to effectively detoxify CN, a deficiency of such amino acids has also been postulated to reduce a key antioxidant, glutathione, thereby hindering responses to the cyanide exposure-related oxidative stress, however, direct evidence of this mechanism has been scarce 12,13

However, numerous studies document higher incidences of mitochondrial abnormalities in affected individuals

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Different standard concentrations and diluted plasma (1:5) were preincubated with labeled anti-MG trace antibody in U-shaped microtiter plate at room temperature