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The programmed cell death expresses morphology characterized by reduction or absence of the mitochondrial crista, rupture of the outer membrane, decreased mitochondrial volume and increased membrane density (Baiskhanova and Schfer 2024)

It works primarily by binding to the ghrelin receptor (GHS-R1a)

Modern methods of 16S rRNA sequencing and metagenomic analysis provide high-resolution insights into the microbiota, allowing the identification of specific profiles associated with diabetes and enabling the development of personalized dysbiosis-correction strategies