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In contrast to the digestive tract, the significance of facilitated or active transport mechanisms in the oral mucosa is believed to be low

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Functional investigations in mouse models have demonstrated that pharmacological suppression of MEK1/2, the direct upstream activator of ERK1/2, significantly attenuates airway inflammation, hyperresponsiveness, and key remodeling characteristics (Liu et al

The quinone metabolites of ISO that form superoxide anions and other reactive oxygen species (ROS) when they react with oxygen might cause oxidative stress by interfering with antioxidant enzymes [124]