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These results suggest that GAPDH inhibition-induced NET formation is not caused by a general reduction in glycolysis rate, loss of ATP, or loss of glycolytic end products, but specifically by a block at the level of GAPDH

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Neuroprotective effect and mechanism of action of tetramethylpyrazine nitrone for ischemic stroke therapy

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also arrived at similar conclusions and revealed the correlation between histone succinylation and tumor progression [80, 83]

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Iron chelation by deferoxamine prevents renal interstitial fibrosis in mice with unilateral ureteral obstruction

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