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The homocysteine-inducible, endoplasmic reticulum (ER) stress-inducible, ubiquitin-like domain member 2 (HERPUD1) in cultured HELA cells facilitate cytoprotective effects against oxidative stress by suppressing the inositol 1,4,5-triphosphate receptor and reducing the transfer of calcium ions from the endoplasmic reticulum to mitochondria [41]

Additionally, concerns regarding long-term biosafety, redox imbalance, metal accumulation, and the scalability of complex nanomaterial synthesis limit regulatory progress

Minor side effects are usually related to the IV process itself, such as minor discomfort at the insertion site or a temporary metallic taste during the infusion

Additionally, SLC1A5 plays a critical role in the polarization and inflammatory activity of TH1 and TH17 cells (124)