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Description

The neuropathology of autism: defects of neurogenesis and neuronal migration, and dysplastic changes

Discussion L1 GUS distribution across the SSN points to distinct substrate specificities We previously demonstrated that a small set of L1 GUS enzymes are capable of processing small glucuronide substrates and are susceptible to selective GUS inhibition, while non-L1 GUS enzymes process larger glucuronide-containing substrates and are not effectively inhibited 20,21