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doi: 10.3389/fnmol.2017.00033 Received 29 September 2016 Accepted 27 January 2017 Published 13 February 2017 Volume 10 - 2017 Edited by Ildik Rcz, University of Bonn, Germany Reviewed by Wei Chen, Beijing Institute of Pharmacology and Toxicology, China

Compared with non-smokers, primary ATII cells from patients with COPD exhibited diverse structural changes, such as reduced mitochondrial area and perimeter, and impaired mitochondrial cristae arrangement

Activating SIRT3 in adipocytes could be a potential strategy to alleviate obesity-related metabolic diseases

This effect was accompanied by a significant reduction in released cytosolic proteins such as myoglobin and creatine kinase [50, 52, 53] as well as attenuation in plasma marker of oxidative stress - malondialdehyde [51, 53, 54]