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Consequently, ectopic overexpression of NPLOC4 reverses CuET-mediated cytotoxicity (Skrott et al., 2017), although the role of VCP in this process remains unknown

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3) GPX4-independent ferroptosis suppressionGCH1BH4 pathway: GTP cyclohydrolase I (GCH1) and its metabolite tetrahydrobiopterin/dihydrobiopterin (BH4/BH2) protect cells from ferroptosis by selectively inhibiting the consumption of phospholipids containing two polyunsaturated acyl chains (105, 106)

The same year, a team featuring some of the same researchers Early clinical studies on AP39 have only been done in animals