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Key questions remain regarding its ability to effectively inhibit NLRP3 inflammasome activation within the complex milieu of diabetic lung injury, and whether long-term suppression of this core innate immune pathway might lead to the activation of compensatory inflammatory pathways or other unknown off-target effects, all of which require longer-term clinical investigation

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Observations indicated increases in cortical BMC and total bone volume, likely due to enhanced bone dimensions rather than volumetric mineral density changes

Taken together, these preclinical data support interventions that target UA to counteract oxidative stress in the ischemic penumbra and protect endothelial cell function after cerebral ischemia-reperfusion injury