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Conclusion Mild to moderate hyperhomocysteinemia seems to be an independent and causal risk factor for atherosclerotic vascular disease

These findings not only deepen our understanding of CD pathophysiology but also position CEAAs, Cys, and Tyr as promising candidates for therapeutic targeting and biomarker development

Recently, Michael Millers group at the University of Alabama showed that pheromone-sensing neurons in the C

Osteoclast differentiation factor Denosumab (DMab) is a human monoclonal IgG2 antibody that inhibits bone resorption by binding and inhibiting the receptor activator of the NF-kB ligand (RANKL), an essential cytokine for osteoclastogenesis and bone resorption