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In a second step the gene coding for human NACB isoform 2 preceded by a ribosome binding site was cloned into the same vector using EcoRI and NotI

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In this same study, the analysis of the influence of the deletion of and on clinical and biochemical changes did not indicate a significant association, and this suggests that the null polymorphism may be associated with the risk of developing the disease, but not with the biochemical alterations analyzed

[4] [7] Its identity in the literature is a single, well-characterized molecule with a defined approval history