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Although our previous study of D2R-DAT interaction was conducted in mouse tissues, we have confirmed the existence of D2R-DAT interaction using rat striatal tissue and that TAT-DAT NT peptide was able to disrupt the D2R-DAT interaction in this study (Fig

One study indicated that down-regulating the expression of the pro-apoptotic factor Bax and reducing the Bax/Bcl-2 ratio effectively mitigated degenerative lesions in dopaminergic neurons associated with PD (Yang et al., 2025)

This mechanism works via the following pathway: polyphenols generate H 2 O 2 in the blood system in the endothelial cell membrane, thereby activating signaling factors

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