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Discussion Despite the evolution of EGFR mutation-targeting tyrosine kinase inhibitors (TKIs) through several generations, clinical challenges persist, including primary and acquired resistance, as well as a lack of response to therapy in EGFR wild-type cases

, etc., have significant vasodilatory bioactivities in vitro , but the hypotensive efffect of only 40% of TMPs have been investigated in vivo at present, such as G

Erastin, a ferroptosis inducer, binds to VDAC2 and VDAC3 on the outer mitochondrial membrane, inducing mitochondrial dysfunction and oxidant release, ultimately triggering ferroptosis in tumor cells