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GHRP-2 has an extensive published research profile spanning more than three decades, with peer-reviewed literature covering GH axis pharmacology, pituitary biology, cardioprotection, HPA axis interaction, and comparative GHRP pharmacology

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These findings suggest that MPLA reprograms macrophage antimicrobial function through alternative, NO-independent mechanisms, likely dominated by ROS-mediated oxidative stress and metabolic reprogramming, rather than canonial nitrogen-centered cytotoxic pathways