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Ser is a substrate for mitochondrial onecarbon metabolism, which generates NADPH while converting Ser + tetrahydrofolate (THF) into glycine (Gly) + THF + formate through the activity of serine hydroxymethyl transferase 2 (SHMT2), methyleneTHF dehydrogenase (MTHFD) 2, and MTHFD1L

Linus Pauling Institute Oregon State University Updated in December 2016 by: Barbara Delage, Ph.D

The Phase 2 trial demonstrated that gradual dose titration reduced the incidence of GI adverse events compared to starting at the target dose directly

Senescence was induced with 100 nM 4-OHT for 7 days, while non-senescent cell controls were obtained by treating cells with DMSO