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Importantly, ferroptosis is inherently pro-inflammatory, establishing a self-amplifying pathogenic axis that operates across major cardiovascular diseases

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In general, glycolytic metabolites upstream of GAPDH were enriched upon high dose VitC treatment, while those downstream were depleted, in line with an inhibition of GAPDH by VitC, ultimately leading to the disruption of glycolysis and TCA cycle also observed in several proteomics studies (Table 4, Fig