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Potentially as a consequence of the heterogeneity of functionally redundant cell signaling networks and multiple genetic alterations 43 , glioblastoma remains largely refractory to current approaches of cancer therapy targeting specific signaling pathways 44

The tricarboxylic acid (TCA) cycle, glycolysis, fatty acid metabolism, and nucleotide synthesis are key metabolic pathways that maintain intracellular homeostasis and regulate DNA repair mechanisms

Var17 and Var18 were upstream of the long intergenic non-coding element, LINC02228 , while Var19 was intronic to it (Fig

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