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Despite the null results on neuroplasticity, the observed decrease in corticospinal excitability and inhibition in the treatment group might suggest improved synaptic efficiency, warranting further investigation over longer intervention periods or with higher doses ( 3.3 Antioxidant and anti-inflammatory properties Oxidative stress and chronic low-grade inflammation are central features of brain aging and contribute to neuronal damage, reduced synaptic plasticity, and cognitive decline ( In rodent models, aging is consistently linked to cognitive deficits and heightened inflammation

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These studies highlight the central role of mitochondrial oxidative stress, lipid peroxidation, and ferroptosis in renal IRI pathology