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An increase in tonus of 0.5 mN was used as threshold for vasoconstriction because this difference is larger than the variation observed in the basal tonus of aorta rings

Thus, the transition from crystal deposition to acute inflammation can be conceptualized as a two-hit process: First hit: MSU crystal deposition on cartilage surfaces activates synovial fibroblasts to release CCL2/MCP-1, recruiting monocytes and establishing subclinical inflammation

Hypoxia-inducible factor activation in diabetic kidney disease

The protocol was evaluated only up to the germination stage, and conversion into complete plantlets remained limited, with abnormalities frequently observed