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This mechanism promotes early satiety signals and reduces the rate of nutrient absorption, leading to flattened postprandial glucose excursions and reduced caloric intake

The mechanism of action, involving natural beta-3 adrenergic receptors and lipolytic pathways, suggests that chronic use is unlikely to produce novel safety concerns beyond what was observed in clinical trials

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Limitations Follow-up studies should examine the same rating scales and biomarkers at later time points, including 6 months, 1 and 2 years after the acute infection, to delineate which patients show protracted chronic fatigue syndrome and GAD (which tend to be chronic disorders) and remitted depression (which most often comes in short-lasting episodes) [54, 105]