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While transient activation supports DNA repair, PARP hyperactivation leads to excessive NAD + and ATP consumption, resulting in metabolic collapse, mitochondrial dysfunction, and cell death by parthanatos, necrosis, or apoptosis under conditions of oxidative stress and ischemia/reperfusion (I/R) injury [139, 204]

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This form of glutathione is particularly effective for those looking to maximize their supplementation

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