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*Correspondence: Eraci Drehmer, [email protected] This article was submitted to Oxidant Physiology, a section of the journal Frontiers in Physiology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Funding This research was funded by the National Natural Science Foundation of China (82102701, 81971864), Sino-German grant (M-0603 CDZ), and the Kln Fortune Program/Faculty of Medicine (Nr

doi: 10.1097/ccm.0b013e3182546855 297 ZinovkinR

Mechanistically, these effects appeared to be mediated through mitochondrial uncoupling, AMPK activation, modulation of mTOR and PPAR signaling, improved mitochondrial bioenergetics, antioxidant defense and global metabolic reprogramming (Fig