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Focal ischemia followed by chronic hypoperfusion triggers changes in AQP4 distribution from perivascular to parenchymal and increases amyloid- deposits which contributes to PSCI ( Permeability of the scar-border allows the accumulation of antibodies in the parenchyma after stroke (Zbesko et al., 2018), which is linked to the development of PSCI ( Neuroinflammation and oxidative stress linked to secondary neurodegeneration after ischemic stroke is proposed to drive the development of PSCI

Subcellular localization experiments indicated that the AsGSTU15 protein is located in both the cytoplasm and nucleus (Fig

The present study was supported by the Scientific Grant Agency of the Ministry of Education, Science, Research and Sport of the Slovak Republic (Bratislava, Slovak Republic