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Employing a qualitative, multi-method approach, the research integrates interviews, policy document analysis, and a review of European projects

In populations with schizophrenia, dysfunction in glutamate metabolism and decreased glutamate levels in the pre-frontal cortex have been reported.68 The addition of cysteine has been shown to modulate glutamate levels through glutamatecystine exchange, and GSH has been shown to modulate the binding of glutamate to N -methyl-d-aspartate receptors.69 N -acetylcysteine may be beneficial in the treatment of schizophrenia by targeting both oxidative stress and glutamatergic dysfunction, suggesting that the phenotype is a result of interactions of multiple neurotransmitter pathways70 that interact with oxidative and inflammatory systems, which are additionally implicated in the disorder

It is essential for shielding cells from oxidative damage and maintaining immunological function