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Significant progress in the development of analytical methods in the last two or three decades has made it possible to quantify the degree of oxidative damage in various pathological tissues (Gulcin 2020)

The interaction between aPL and targets like 2GPI triggers the activation of endothelial cells, platelets, monocytes, and neutrophils, as well as the complement and coagulation systems, forming a key molecular mechanism underlying APS pathogenesis ( 5 Limitations This study has several limitations

Neutrophils are the short lived, fugitive, most abundant and terminally differentiated innate immune cells, eliminate infections through evolutionary conserved biological processes such as phagocytosis, degranulation, producing extracellular traps and regulating macrophages and B cell functions [3]