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11, 2342 (2012), Abstract Full Text Mitochondrial metabolism in Parkinsons disease impairs quality control autophagy by hampering microtubule-dependent traffic: D.M

Accordingly, a multitude of molecules and antibodies to interact either with NF-B and C/EBP transcriptional activities at different levels have been developed (summarized in: 81)

Interestingly, FAT10 does not contain any disordered segments long enough for proteasomal engagement and its degradation in vivo is not blocked by p97 inhibitors 33 , leaving the question of how it can bypass the requirement for a bipartite degradation signal