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[PubMed] Hare, W.A., E

Hyperglycemia is hypothesized to induce myocardial lipid peroxidation via mitochondrial iron overload and suppression of glutathione peroxidase 4 (GPX4) activity, leading to impaired myocardial contractile function ( Over recent decades, extensive research has elucidated DCM pathophysiology, with mounting evidence implicating impaired mitochondrial energy metabolism as a central pathogenic mechanism ( 4 Therapeutic interventions targeting cardiac energy metabolism In response to the pathological features of cardiac metabolic dysregulation, multifaceted intervention strategies have been developed in both clinical and research settings