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Minkler et al

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We hypothesized that low glutathione redox status would be associated with elevated markers of oxidative protein and DNA damage, inflammation and mitochondrial superoxide production in two regions that have been reported to be abnormal in autism, cerebellum and BA22

2,6,7 This discrepancy may be attributed to possible contamination of the antibiotics used in an earlier era or to differences in age, ethnicity, and genetics in study participants