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van de Veen, W

Similarly, ALX/FPR2, which recognizes both lipoxin A4 (LXA4) and RvD1, can be activated by serum amyloid A, triggering the phosphoinositide-3 kinase (PI3K) and nuclear factor-B (NF-B) signaling pathways and promoting inflammation [32]

The administration of Wilforlide A reduced clinical scores, joint swelling and histological damage of collagen-induced RA mice by inhibiting the secretion of pro-inflammatory factors (MCP1, GM-CSF and M-CSF) and iNOS in the synovium (140)

However, the underlying molecular mechanism of ferroptosis in AD, when ferroptosis occurs in the disease course, and which ferroptosis-related genes are differentially expressed in AD remains to be established