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These data suggest that erastin treatment can induce two opposing effects on SLC7A11 transcription: transcriptional activation (through a still unknown mechanism) and transcriptional repression (through decreasing H2Bub occupancy on the SLC7A11 promoter (Wang et al., 2019b) and potentially activating ATF3 (Wang et al., 2020a)]

A 109 , 93939401 (2005)

Advancements in single-cell sequencing technologies have played a pivotal role in advancing our understanding of the cancer cell-of-origin hypothesis

For example, patients with autoimmune diseases may experience symptom alleviation through interventions targeting neutrophil activity regulation