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Description

161 Th17 cells are excreted from the intestine in an S1PR1-dependent manner and subsequently migrate to the kidney via the CCL20/C-C motif chemokine receptor 6 (CCR6) axis, thereby causing nephritis

Busulfan metabolism was modeled as a GSH-dependent conjugation process, with the scaling parameter S GSH quantifying the proportionality between busulfan metabolism and corresponding GSH depletion (Equation 2).Where represents the amount of busulfan in the central compartment, represents the amount of GSH in the theoretical compartment, represents the central volume of distribution, and represents the busulfan elimination constant

Sulfiredoxin protein is critical for redox balance and survival of cells exposed to low steady-state levels of H 2 O 2

Glutathione conjugation protects against AQ-induced toxicity and GSTP1 is able to conjugate its quinoneimine metabolites AQ-QI and DEA-QI with glutathione