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In summary, the multi-pathway impact of CAT activity regulated by GSNOR under Cd stress, included the regulation of NO accumulation, modification of the S -nitrosylation level in the CAT protein and changing expression of CAT2 in shoots (Fig

Experimental studies in neuropathic pain models suggest that tDCS exerts anti-inflammatory and neuroprotective effects, including the down-regulation of pro-inflammatory cytokines (e.g., TNF- , IL-1), the up-regulation of IL-10, and the suppression of microglial and astrocytic activation, thereby fostering adaptive plasticity and reducing hypersensitivity (85, 86)

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