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This tripeptide combines cysteine, glycine, and glutamic acid, and exists at surprisingly high levels5 millimolarin most cells

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Senescent tumor cells, driven by replicative exhaustion or stress-induced DNA damage, undergo irreversible cell cycle arrest and secrete senescence-associated secretory phenotypes (SASP) that paradoxically suppress tumorigenesis but promote TME remodeling [7, 10, 15, 16, 21]

90 All these results show that signaling through the PSD95/nNOS complex is crucial for excitotoxicity in ischemic stroke and contributes to the neurotoxic effects of extra-synaptic NMDARs