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Abscence of Ph+ cells in CD34- lin- SP bone marrow progenitor cells from patients with chronic myeloid leukemia (CML)

Although most of these data were generated in regenerative settings rather than during steady-state aging, they support a model in which aging can reduce the MuSC pool through probabilistic loss or dysfunction of MuSCs (or labeled lineages) without implying deterministic, subset-specific collapse of heterogeneity (Fig

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"GHK influenced the expression of about 4,000 human genes, shifting their expression from a diseased or aged state toward a healthier profile." Pickart et al., BioMed Res Int (2014) Loading, Tapering, and Continuous Use# There is no loading phase