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Aft1 was constitutively localized in Grx3 and Grx4, iron homeostasis was disrupted, and they became hypersensitive to oxidative stress

Following injury, a complex inflammatory response is triggered

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These single-cell expression profiles systematically delineate the spatiotemporal regulation patterns of ferroptosis-suppressing signature genes within the renal disease microenvironment, providing direct cellular and molecular evidence for elucidating the mechanisms of ferroptosis imbalance in AKD and CKD pathogenesis, while offering important references for subsequent research on intervention strategies targeting ferroptosis-suppressing pathways in kidney diseases