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Administration of low concentrations of luteolin (15 M) resulted in enhanced cell viability, inhibition of lactate dehydrogenase (LDH) release, and a reduction in levels of IL-1, concomitantly modulating the expression of genes pertinent to PD: specifically, an elevation in Park2, a reduction in Lrrk2, and an enhancement of Nrf2 and Trx1 expression

Later in the infection, the combination of signals and pathways induced for bacterial internalization may promote a localized loss of barrier function, allowing the paracellular route to occur, making bacterial internalization a requisite step for further invasion

*Correspondence: Xiao-Hong Sun, [email protected] This article was submitted to Parkinsons Disease and Aging-related Movement Disorders, a section of the journal Frontiers in Aging Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers