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Cytoplasmic GSDMD is cleaved by Caspase-1, resulting in the release of its N-terminal domain from the autoinhibitory conformation binding to its C-terminal domain

EAAC1-deficient mice showed age-dependent brain atrophy, learning/memory dysfunction, and reduced brain GSH levels [108]

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demonstrated that the use of structure-based design of a self-assembling protein nanoparticle presenting a pre-fusion-stabilized DS-Cav1 in a repetitive array on the nanoparticle exterior induced neutralizing antibody responses up to tenfold higher than trimeric DS-Cav1 alone 260