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Bcma is essential for the survival of long-lived bone marrow plasma cells

HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use

Development of erythroid and myeloid progenitors in the yolk sac and embryo proper of the mouse

Many spotted fever group Rickettsia associate with leukocytes throughout their infectious lifecycles, including macrophages and neutrophils in the skin and in internal organs upon dissemination (7, 2629)