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Pathogenesis May arise from fixed macrophages (Kupffer cells) attached to the sinusoidal endothelial cells or from circulating macrophages, unless metastasized from other organs (e.g., skin, uterus)

Picciotto et al., 2015) as well as elevation of cortisol (Walker et al., 1990), which in turn is associated with IOP elevation (Schwartz and Seddon, 1981) and vascular dysregulation with relevance to Flammer syndrome or endothelial dysfunction-mediated glaucoma (Buckley et al., 2002

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This, in turn, promotes NCOA4-mediated ferritinophagy, resulting in elevated intracellular iron levels and lipid peroxidation, ultimately triggering ferroptosis and contributing to osteoporotic pathology