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Importantly, the observed oxidative damage and mitochondrial impairment following GCLC knockdown were reproduced across multiple assays and time points, reinforcing its critical role in redox regulation and cellular viability [31,32,33]

Circulating RBCs are subject to a continuous onslaught of endogenous and exogenous oxidative stress, and expectedly more stress is incurred during storage and during the harsh treatment by chemicals and/or exposure to UV lights [35]

These phenomena are largely impacted by post-donation hemoglobin- oxygen saturation, which has been noted to vary widely across healthy blood donors

Anti-inflammatory effects of spermidine in lipopolysaccharide-stimulated BV2 microglial cells